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Correction: Moesin silencing enhances the radiosensitivity of glioblastoma by inhibiting DNA damage repair via CD44/AKT1 signaling pathway

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Published in:Discover Oncology
Format: Online Article RSS Article
Published: 2026
Subjects:
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container_title Discover Oncology
description
discipline_display Endocrinology
discipline_facet Endocrinology
format Online Article
RSS Article
genre Journal Article
id rss_article:106755
institution FRELIP
journal_source_facet Discover Oncology
last_indexed 2026-07-26T02:01:08.348Z
publishDate 2026
publishDateSort 2026
record_format rss_article
spellingShingle Correction: Moesin silencing enhances the radiosensitivity of glioblastoma by inhibiting DNA damage repair via CD44/AKT1 signaling pathway
Endocrinology
General
Endocrinology
sub_discipline_display General
sub_discipline_facet General
subject_display Endocrinology
General
Endocrinology
Endocrinology
General
Endocrinology
subject_facet Endocrinology
General
Endocrinology
title Correction: Moesin silencing enhances the radiosensitivity of glioblastoma by inhibiting DNA damage repair via CD44/AKT1 signaling pathway
title_auth Correction: Moesin silencing enhances the radiosensitivity of glioblastoma by inhibiting DNA damage repair via CD44/AKT1 signaling pathway
title_full Correction: Moesin silencing enhances the radiosensitivity of glioblastoma by inhibiting DNA damage repair via CD44/AKT1 signaling pathway
title_fullStr Correction: Moesin silencing enhances the radiosensitivity of glioblastoma by inhibiting DNA damage repair via CD44/AKT1 signaling pathway
title_full_unstemmed Correction: Moesin silencing enhances the radiosensitivity of glioblastoma by inhibiting DNA damage repair via CD44/AKT1 signaling pathway
title_short Correction: Moesin silencing enhances the radiosensitivity of glioblastoma by inhibiting DNA damage repair via CD44/AKT1 signaling pathway
title_sort correction: moesin silencing enhances the radiosensitivity of glioblastoma by inhibiting dna damage repair via cd44/akt1 signaling pathway
topic Endocrinology
General
Endocrinology
url https://link.springer.com/article/10.1007/s12672-026-05577-x