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The interferon-stimulated gene product HERC5 inhibits human LINE-1 retrotransposition with an ISGylation-independent mechanism

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Published in:Nucleic Acids Research
Format: Online Article RSS Article
Published: 2026
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container_title Nucleic Acids Research
description
discipline_display Natural Sciences
discipline_facet Natural Sciences
format Online Article
RSS Article
genre Journal Article
id rss_article:47310
institution FRELIP
journal_source_facet Nucleic Acids Research
publishDate 2026
publishDateSort 2026
record_format rss_article
spellingShingle The interferon-stimulated gene product HERC5 inhibits human LINE-1 retrotransposition with an ISGylation-independent mechanism
Biology
Natural Sciences — Life Sciences
Natural Sciences
sub_discipline_display Natural Sciences — Life Sciences
sub_discipline_facet Natural Sciences — Life Sciences
subject_display Biology
Natural Sciences — Life Sciences
Natural Sciences
Biology
Natural Sciences — Life Sciences
Natural Sciences
subject_facet Biology
Natural Sciences — Life Sciences
Natural Sciences
title The interferon-stimulated gene product HERC5 inhibits human LINE-1 retrotransposition with an ISGylation-independent mechanism
title_auth The interferon-stimulated gene product HERC5 inhibits human LINE-1 retrotransposition with an ISGylation-independent mechanism
title_full The interferon-stimulated gene product HERC5 inhibits human LINE-1 retrotransposition with an ISGylation-independent mechanism
title_fullStr The interferon-stimulated gene product HERC5 inhibits human LINE-1 retrotransposition with an ISGylation-independent mechanism
title_full_unstemmed The interferon-stimulated gene product HERC5 inhibits human LINE-1 retrotransposition with an ISGylation-independent mechanism
title_short The interferon-stimulated gene product HERC5 inhibits human LINE-1 retrotransposition with an ISGylation-independent mechanism
title_sort the interferon-stimulated gene product herc5 inhibits human line-1 retrotransposition with an isgylation-independent mechanism
topic Biology
Natural Sciences — Life Sciences
Natural Sciences
url https://academic.oup.com/nar/article/doi/10.1093/nar/gkag334/8664513?rss=1