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A LINE-1 retrotransposon promotes SEMA3C expression as a -regulatory enhancer to sustain breast cancer stem cell survival

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Published in:Science China Life Sciences
Format: Online Article RSS Article
Published: 2026
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container_title Science China Life Sciences
description
discipline_display Natural Sciences
discipline_facet Natural Sciences
format Online Article
RSS Article
genre Journal Article
id rss_article:47356
institution FRELIP
journal_source_facet Science China Life Sciences
publishDate 2026
publishDateSort 2026
record_format rss_article
spellingShingle A LINE-1 retrotransposon promotes SEMA3C expression as a -regulatory enhancer to sustain breast cancer stem cell survival
Biology
Natural Sciences — Life Sciences
Natural Sciences
sub_discipline_display Natural Sciences — Life Sciences
sub_discipline_facet Natural Sciences — Life Sciences
subject_display Biology
Natural Sciences — Life Sciences
Natural Sciences
Biology
Natural Sciences — Life Sciences
Natural Sciences
subject_facet Biology
Natural Sciences — Life Sciences
Natural Sciences
title A LINE-1 retrotransposon promotes SEMA3C expression as a -regulatory enhancer to sustain breast cancer stem cell survival
title_auth A LINE-1 retrotransposon promotes SEMA3C expression as a -regulatory enhancer to sustain breast cancer stem cell survival
title_full A LINE-1 retrotransposon promotes SEMA3C expression as a -regulatory enhancer to sustain breast cancer stem cell survival
title_fullStr A LINE-1 retrotransposon promotes SEMA3C expression as a -regulatory enhancer to sustain breast cancer stem cell survival
title_full_unstemmed A LINE-1 retrotransposon promotes SEMA3C expression as a -regulatory enhancer to sustain breast cancer stem cell survival
title_short A LINE-1 retrotransposon promotes SEMA3C expression as a -regulatory enhancer to sustain breast cancer stem cell survival
title_sort a line-1 retrotransposon promotes sema3c expression as a -regulatory enhancer to sustain breast cancer stem cell survival
topic Biology
Natural Sciences — Life Sciences
Natural Sciences
url https://link.springer.com/article/10.1007/s11427-025-3210-8