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Correction: The disruptor of telomeric silencing 1-like (DOT1L) promotes peritoneal fibrosis through the upregulation and activation of protein tyrosine kinases

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Published in:Molecular Biomedicine
Format: Online Article RSS Article
Published: 2026
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container_title Molecular Biomedicine
description
discipline_display Biology
discipline_facet Biology
format Online Article
RSS Article
genre Journal Article
id rss_article:82251
institution FRELIP
journal_source_facet Molecular Biomedicine
publishDate 2026
publishDateSort 2026
record_format rss_article
spellingShingle Correction: The disruptor of telomeric silencing 1-like (DOT1L) promotes peritoneal fibrosis through the upregulation and activation of protein tyrosine kinases
Biology
General
Biology
sub_discipline_display General
sub_discipline_facet General
subject_display Biology
General
Biology
Biology
General
Biology
subject_facet Biology
General
Biology
title Correction: The disruptor of telomeric silencing 1-like (DOT1L) promotes peritoneal fibrosis through the upregulation and activation of protein tyrosine kinases
title_auth Correction: The disruptor of telomeric silencing 1-like (DOT1L) promotes peritoneal fibrosis through the upregulation and activation of protein tyrosine kinases
title_full Correction: The disruptor of telomeric silencing 1-like (DOT1L) promotes peritoneal fibrosis through the upregulation and activation of protein tyrosine kinases
title_fullStr Correction: The disruptor of telomeric silencing 1-like (DOT1L) promotes peritoneal fibrosis through the upregulation and activation of protein tyrosine kinases
title_full_unstemmed Correction: The disruptor of telomeric silencing 1-like (DOT1L) promotes peritoneal fibrosis through the upregulation and activation of protein tyrosine kinases
title_short Correction: The disruptor of telomeric silencing 1-like (DOT1L) promotes peritoneal fibrosis through the upregulation and activation of protein tyrosine kinases
title_sort correction: the disruptor of telomeric silencing 1-like (dot1l) promotes peritoneal fibrosis through the upregulation and activation of protein tyrosine kinases
topic Biology
General
Biology
url https://link.springer.com/article/10.1186/s43556-026-00484-7