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The ubiquitin E3 ligase TRIM59 in myeloid cell regulates complement C3 through degrading CCAAT/enhancer binding protein β to promote neuron survival after ischemic stroke

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Published in:Cell & Bioscience
Format: Online Article RSS Article
Published: 2026
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container_title Cell & Bioscience
description
discipline_display Bioengineering
discipline_facet Bioengineering
format Online Article
RSS Article
genre Journal Article
id rss_article:98095
institution FRELIP
journal_source_facet Cell & Bioscience
last_indexed 2026-07-01T03:40:58.034Z
publishDate 2026
publishDateSort 2026
record_format rss_article
spellingShingle The ubiquitin E3 ligase TRIM59 in myeloid cell regulates complement C3 through degrading CCAAT/enhancer binding protein β to promote neuron survival after ischemic stroke
Bioengineering
General
Bioengineering
sub_discipline_display General
sub_discipline_facet General
subject_display Bioengineering
General
Bioengineering
subject_facet Bioengineering
General
Bioengineering
title The ubiquitin E3 ligase TRIM59 in myeloid cell regulates complement C3 through degrading CCAAT/enhancer binding protein β to promote neuron survival after ischemic stroke
title_alt La ubiquitina E3 ligasa TRIM59 en células mieloides regula el complemento C3 mediante la degradación de la proteína de unión a CCAAT/ potenciador beta para promover la supervivencia neuronal después de un accidente cerebrovascular isquémico
L'ubiquitine ligase E3 TRIM59 dans les cellules myéloïdes régule le complément C3 en dégradant la protéine de liaison CCAAT/enhancer bêta pour favoriser la survie neuronale après un accident vasculaire cérébral ischémique
A ubiquitina E3 ligase TRIM59 em células mieloides regula o complemento C3 através da degradação da proteína de ligação ao intensificador CCAAT/β para promover a sobrevivência neuronal após acidente vascular cerebral isquêmico
title_auth The ubiquitin E3 ligase TRIM59 in myeloid cell regulates complement C3 through degrading CCAAT/enhancer binding protein β to promote neuron survival after ischemic stroke
title_es_txt La ubiquitina E3 ligasa TRIM59 en células mieloides regula el complemento C3 mediante la degradación de la proteína de unión a CCAAT/ potenciador beta para promover la supervivencia neuronal después de un accidente cerebrovascular isquémico
title_fr_txt L'ubiquitine ligase E3 TRIM59 dans les cellules myéloïdes régule le complément C3 en dégradant la protéine de liaison CCAAT/enhancer bêta pour favoriser la survie neuronale après un accident vasculaire cérébral ischémique
title_full The ubiquitin E3 ligase TRIM59 in myeloid cell regulates complement C3 through degrading CCAAT/enhancer binding protein β to promote neuron survival after ischemic stroke
title_fullStr The ubiquitin E3 ligase TRIM59 in myeloid cell regulates complement C3 through degrading CCAAT/enhancer binding protein β to promote neuron survival after ischemic stroke
title_full_unstemmed The ubiquitin E3 ligase TRIM59 in myeloid cell regulates complement C3 through degrading CCAAT/enhancer binding protein β to promote neuron survival after ischemic stroke
title_pt_txt A ubiquitina E3 ligase TRIM59 em células mieloides regula o complemento C3 através da degradação da proteína de ligação ao intensificador CCAAT/β para promover a sobrevivência neuronal após acidente vascular cerebral isquêmico
title_short The ubiquitin E3 ligase TRIM59 in myeloid cell regulates complement C3 through degrading CCAAT/enhancer binding protein β to promote neuron survival after ischemic stroke
title_sort the ubiquitin e3 ligase trim59 in myeloid cell regulates complement c3 through degrading ccaat/enhancer binding protein β to promote neuron survival after ischemic stroke
topic Bioengineering
General
Bioengineering
url https://link.springer.com/article/10.1186/s13578-026-01605-z